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Santa Cruz Biotechnology
glutamate decarboxylase gad67  Glutamate Decarboxylase Gad67, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 88/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more https://www.bioz.com/product/gad67+activity/GAD-67+Lentiviral+Activation+Particles/pm30048986-94-51-54 Average 88 stars, based on 1 article reviews
glutamate decarboxylase gad67 - by Bioz Stars,
2026-09
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CRISPR/Cas9 KO Plasmids consists of GAD-67-specific 20 nt guide RNA sequences derived from the GeCKO (v2) library. For CRISPR gene knockout, gRNA sequences direct the Cas9 protein to induce a site-specific double strand break (DSB)
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GAD1 CRISPRa kit CRISPR gene activation of human glutamate decarboxylase 1
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Journal: Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology
Article Title: Exercise Training Attenuates Proinflammatory Cytokines, Oxidative Stress and Modulates Neurotransmitters in the Rostral Ventrolateral Medulla of Salt-Induced Hypertensive Rats.
doi: 10.1159/000492095
Figure Lengend Snippet: Fig. 5. Effects of exercise training on the 67-kDa isoform of glutamate decarboxylase (GAD67) immunoreactivity and neuronal activity in the RVLM of NS rats and HS rats. (A and C) A representative immunohistochemistry image and the column diagram showing the effects of exercise training on the positive neurons of GAD67 (brown yellow) in the RVLM of NS rats and HS rats. (B and D) A representative immunohistochemistry image and the column diagram showing the effects of exercise training on the Fra-LI (brown dots, an indicator of chronic neuronal excitation) expression in the RVLM of NS rats and HS rats. Values are mean ± SE. *P<0.05 vs. NS groups; †P<0.05 HS + Ex vs. HS + Sed.
Article Snippet: Published by S. Karger AG, Basel www.karger.com/cpb Li et al.: Exercise Training in Salt-Induced Hypertension Immunohistochemistry and immunofluorescence studies Immunohistochemical labeling was performed in floating sections (18 μm) as previously described [21, 22] to identify Fra-like (Fra-LI, a marker of chronic neuronal activation), tumour necrosis factor-alpha (TNF-α) and 67-kDa isoform of
Techniques: Activity Assay, Immunohistochemistry, Expressing
Journal: Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology
Article Title: Exercise Training Attenuates Proinflammatory Cytokines, Oxidative Stress and Modulates Neurotransmitters in the Rostral Ventrolateral Medulla of Salt-Induced Hypertensive Rats.
doi: 10.1159/000492095
Figure Lengend Snippet: Fig. 7. Protein expression of TNF-α, GAD67, gp91phox and NOX4 in the RVLM and ANP and β-MHC in the heart of NS rats and HS rats. (A) HS rats had higher levels of TNF-α and lower levels of GAD67 in the RVLM when compared with NS rats. Exercise training decreased the protein expression of TNF-α and increased the protein express of GAD67 in the RVLM of HS rats. (B) HS rats had higher levels of gp91phox and NOX4 in the RVLM when compared with NS rats. Exercise training decreased the protein expression of gp91phox and NOX4 in the RVLM when compared with NS rats. (C) Western blot for ANP and β-MHC expressions in the heart were lower in exercise training-treated HS rats than in sedentary HS rats. Values are mean ± SE. *P<0.05 vs. NS groups; †P<0.05 HS + Ex vs. HS + Sed.
Article Snippet: Published by S. Karger AG, Basel www.karger.com/cpb Li et al.: Exercise Training in Salt-Induced Hypertension Immunohistochemistry and immunofluorescence studies Immunohistochemical labeling was performed in floating sections (18 μm) as previously described [21, 22] to identify Fra-like (Fra-LI, a marker of chronic neuronal activation), tumour necrosis factor-alpha (TNF-α) and 67-kDa isoform of
Techniques: Expressing, Western Blot